The impairment of HMGB1 function results in enhanced mitochondrial DNA damage. During the SCA1 mouse product, around-expression with the HMGB1 protein through an released virus vector bearing the HMGB1 gene facilitates maintenance on the mitochondrial DNA injury, ameliorates the neuropathology along with the motor deficits, and extends the lifespan of https://exendin-477654.blogscribble.com/28367980/not-known-details-about-unc6852